（1）国家自然科学基金专项项目：非洲猪瘟病毒感染与致病机制（31941002，2019.07-2022.06），300万，参与；

（2）国家自然科学基金面上项目：猪繁殖与呼吸综合征病毒非结构蛋白11抑制NLRP3炎症小体激活的分子机制（31872448，2019.01-2022.12），61万元，主持；

（3）“十三五” 国家重点研发计划项目子课题：以SPF猪为基础的高致病性猪繁殖与呼吸综合征病毒单因子发病模型的建立及其评价（2017YFD0501600，2016.07-2020.12）, 30万元，主持；

（4）国家自然科学基金青年基金：猪繁殖与呼吸综合征病毒NSP1β与HSP40相互作用抑制I型干扰素的产生及其分子机制（31302163，2014.01-2016.12），25万元，主持。

（1）Jiangnan Li 1, JieSong 1, Li Kang, Li Huang, Shijun Zhou, Liang Hu, Jun Zheng,Changyao Li, Xianfeng Zhang, Xijun He, Dongming Zhao, Zhigao Bu *,hangjiangWeng *. pMGF505-7R determines pathogenicity of African swine fever virusinfection by inhibiting IL-1β and type I IFN production.  PLoS Pathogens, 2021, 17: e1009733.

（2） Jie Song, Li Kang, Ting Li, GaihongZhao, Shijun Zhou, Li Huang, JiangnanLi *, Changjiang Weng. Screening of PRRSV- and ASFV-encoded proteinsinvolved in the inflammatory response using a porcine iGLuc reporter. Journalof Virological Methods, 2020, 285: 113958.

（3） Li Huang 1, Tao Xiong 1,Huibin Yu, Quan Zhang, Kunli Zhang, Changyao Li, Liang Hu, Yuanfeng Zhang,Lijie Zhang, Qinfang Liu, Shengnan Wang, Xijun He, Zhigao Bu, Xuehui Cai,Shangjin Cui *, JiangnanLi *, Changjiang Weng *. Encephalo myocarditis virus 3C protease attenuatestype I interferon production through disrupting the TANK-TBK1-IKKepsilon-IRF3complex. Biochemical Journal, 2017, 474(12): 2051-2065.

（4）Jiangnan Li 1, Shengnan Wang1, Changyao Li, Chunlai Wang, Yonggang Liu, Gang Wang, Xijun He,Liang Hu, Yuanyuan Liu, Mengmeng Cui, Caihong Bi, Zengyu Shao, Xiaojie Wang,Tao Xiong, Xuehui Cai *; Li Huang *, Changjiang Weng *. Secondary Haemophilusparasuis infection enhances highly pathogenic porcine reproductive andrespiratory syndrome virus (HP-PRRSV) infection-mediated inflammatoryresponses. Veterinary Microbiology, 2017, 204: 35-42.

（5）Jiangnan Li 1, Liang Hu 1,Yuanyuan Liu 1, Li Huang, Yang Mu, Xuehui Cai, Changjiang Weng *.DDX19A Senses Viral RNA and Mediates NLRP3-dependent Inflammasome Activation.Journal of immunology, 2015, 195(12): 5732-5749.