(1)    黑龙江省重点研发计划指导类项目、GZ20210010、省科技创新基地项目高等级农业实验动物猪培育、质量控制及检测技术研究、2021/10—2023/09、150万元、在研、主持

(2)    国家自然基金面上项目、32072898、基于肠道菌群-肠肺轴探究乳酸菌协助机体抵抗流感病毒感染的作用机制的研究、2020/01— 2024/12、58万元、在研、骨干

(3)    国家重点研发项目、2017YFD0501605、畜禽疫病防控专用实验动物开发、2017/07— 2021/06、1728万元、在研、骨干

(4)    黑龙江省杰出青年基金、JC2016005、紧密连接蛋白在猪流行性腹泻病毒感染中的作用及机制研究、2016/07— 2019/06、50万元、在研、主持

(5)    国家自然基金面上项目、31572497、嗜中性粒细胞在猪流行性腹泻病毒感染中的作用及机制研究、2016/01— 2019/12、75.6万元、在研、主持

(6)    国家自然基金面上项目、31372416、金属蛋白酶ADAM17对猪繁殖与呼吸综合征病毒感染的抑制作用及机制研究、2014/01— 2017/12、80万元、在研、主持

(7)    黑龙江省自然科学基金、LC201418、猪流行性腹泻病毒诱导细胞凋亡机制的研究、2014/07—2017//06、6万元、在研、主持

(8)    兽医生物技术国家重点实验室自主项目、SKLVB201412、PEDV感染诱导细胞凋亡及其机制研究、2014/01— 2014//12、11万元、已结题、主持

(9)    美国农业部合作项目、3625-32000-108-17S、Parallel monitoring of porcine diseases of economic importance in the United States and China、2013/09—2014/09、5.2 万美元、已结题、主持

(10)中国农业科学院院所长基金、0302013003、ADAM17对PRRSV感染的调节作用研究、2013/01— 2013//12、40万元、已结题、主持

(11)美国National Pork Board项目、NPB#07-108、Identification of Type I Interferon Antagonists of PorcineReproductive and Respiratory System Virus Viral Structural Proteins、2008/10— 2010/09、5.7万美元、已结题、主持

(12)“十三五”国家重点研发项目、2017YFD0501605、畜禽疫病防控专用实验动物开发、2017/01-2020/12、1728万元、在研、骨干

(13)黑龙江省科技攻关项目、YC2015D011、猪病毒性腹泻防控新技术、2015/08—2018/08、200万、在研、骨干

(14)中国农业科学院院所长基金增量项目、2013ZL034、重要动物病毒在巨噬细胞中复制及调控天然免疫反应的研究、2013/01— 2013/12、87 万元、已结题、骨干

(15)兽医生物技术国家重点实验室自主项目、SKLVBP167、天然分子调控猪病毒病感染机制的研究、2012/08— 2012//12、40万元、已结题、主持

(1)    HeZhang, Wenjie Ma, Zhi Sun,Changkang Zhu, Gebremeskel Mamu Werid, Yassein M.Ibrahim, Wenli Zhang,Yu Pan, Dongfang Shi, Hongyan Chen, Yue Wang*. Abundance of Lactobacillusin porcine gut microbiota is closely related to immune response following PRRSVimmunization. Vet Microbiol. 2021. 259:109134. 

(2)    ZhangL, Zhang L, Pan Y, Gao J, Xu Y, Li X, Tian Z, Chen H, Yue Wang*. Downregulationof miR-218 by porcine reproductive and respiratory syndrome virus facilitatesviral replication via inhibition of type I interferon. J Bio Chem. 2021. DOI:10.1016/j.jbc.2021.100683. 

(3)    GaoJ, Pan Y, Xu Y, Zhang W, Zhang L, Li X, Tian Z, Chen H and Yue Wang*. Unveilingthe long non-coding RNA profile of porcine reproductive and respiratorysyndrome virus-infected porcine alveolar macrophages. BMC Genomics.2021. 22:177. 

(4)    XuJ, Zhang L, Xu Y, Zhang H, Gao J, Wang Q, Tian Z, Xuan L, Chen H, Wang Yue*.PP2A Facilitates Porcine Reproductive and Respiratory Syndrome VirusReplication by Deactivating irf3 and Limiting Type I Interferon Production. Viruses.2019. 11(10): 948. 

(5)    YangL#, Xu J#, Guo L, Guo T, Zhang L, Feng L, Chen H, Wang Yue*. Porcine Epidemic DiarrheaVirus-Induced Epidermal Growth Factor Receptor Activation Impairs the AntiviralActivity of Type I Interferon. J. Virol. 2018 Mar 28. 92(8). Pii:e02095-17. 

(6)    Zhang J, GuoJ, Yang L, Xu J, Zhang L, Feng L, Chen H, WangYue*. MetalloproteaseADAM17 regulates porcine epidemic diarrhea virus infection by modifyingaminopeptidase N. Virology. 2018. Apr. 517: 24-29. 

(7)    LuoX, Guo L, Zhang J, Xu Y, Gu W, Feng L, WangYue*. Tight junction protein occludin is a porcineepidemic diarrhea virus entry factor. J Virol. 2017, 91(10). pii: e00202-17.

(8)    ZhangJ, Guo L, Xu Y, Yang L, Shi H, Feng L, WangYue*. Characterization of Porcine Epidemic Diarrhea Virus Infectivity in HumanEmbryonic Kidney Cells. ArchivesVirol. 2017, 162(8):2415-2419. 

(9)    GuoL, Luo X, Li R, Xu Y, Zhang J, Ge J, Bu Z, Feng L, Wang Yue*. Porcine epidemic diarrhea virus infection inhibitsinterferon signaling by targeted degradation of STAT1. J Virol. 2016, 90(18):8281-92.

(10) Guo L, Yu H, Gu W, Luo X, Li R, ZhangJ, Xu Y, Yang L, Shen N, Feng L, Wang Yue*.Autophagy negatively regulates transmissible gastroenteritis virus replication.SciRep. 2016, 6:23864. 

(11) Li R, Guo L, Gu W, Luo X, Zhang J, XuY, Tian Z, Feng L, Wang Yue*. Production of Porcine TNFa by ADAM17-mediated cleavage negatively regulates porcine reproductive and respiratory syndromevirus infection. Immunol Res. 2016, 64(3): 711-20.

(12) Gu W, Guo L, Li R, Niu J, Luo X, ZhangJ, Xu Y, Tian Z, Feng L, Wang Yue*. Elevated plasma soluble CD16 levelsin porcine reproductive and respiratory syndrome virus-infected pigs:correlation with ADAM17-mediated shedding. J Gen Virol. 2016,97(3):632-8. 

(13) Gu W, Guo L, Yu H, Niu J, Huang M, LuoX, Li R, Tian Z, Feng L, Wang Yue*. Involvementof CD16 in antibody-dependent enhancement of porcine reproductive andrespiratory syndrome virus infection. J Gen Virol. 2015, 96(7): 1712-1722.

(14) Guo L, NiuJ, Yu H,GuW, Huang M, Li R, Luo X, Tian Z, FengL,Wang Yue*. Modulation of CD163 expression by metalloprotease ADAM17regulatesporcinereproductive and respiratory syndrome virus entry.J Virol.2014, 88(18): 10448-58.

(15) Wang Yue, Wu J,Newton R, Bahaie NS, Long C, WalcheckB.ADAM17 cleaves CD16b(FcγRIIIb) inhumanneutrophils. BBA Mol CellRes. 2013, 1833(3): 680-5

(16) Wang Yue, Robertson JD,andWalcheckB. Differentsignaling pathways stimulate a disintegrin and metalloprotease-17 (ADAM17) in neutrophils during apoptosis and activation. J BioChem. 2011, 286(45):38980-8.

(17) Wang Yue, Zhang A,NiZ, Herrera AH, and Walcheck B. ADAM17activity and other mechanismsof soluble L-selectinproduction during deathreceptor-inducedleukocyte apoptosis.JImmunol. 2010, 184(8): 4447-54.

(18) Wang Yue, Herrera A, Li Y, Belani KK, Walcheck B.Regulation of mature ADAM17byredox agents for L-selectinshedding. J Immunol. 2009,182(4): 2449-2457